Review the molecular mechanisms used by adult schistosoma worms to Essay

Review the molecular mechanisms used by adult schistosoma worms to expire in the bloodstream - Essay ExampleThe remaining eggs often go into circulation and get filtered in the periportal tracts within the liver, and this causes periportal fibrosis. According to Ashton and Wilson (2001), the major schistosome species that motivate individuals are Schistosoma haematobium, Schistosoma mansoni and Schistosoma japonicum. These parasites experience remarkable physiological and morphological changes throughout their life as a means of survival and adaptation to their varying living conditions in different hosts. These parasites are unique because they exhibit unique adaptations both to free-living, as well as parasitic living. The adaptations exhibited by these parasites throw overboard movement between intermediary hosts and the ultimate host. Schistosomes extend within the host by adopting mechanisms that takings the effect of the hosts defense mechanisms. These adaptations occur both at the molecular and structural levels. Schistosomes have various adaptations at the molecular level and these include anti-oxidant production and glycoproteins secretion among others. Structural adaptations also help facilitate survival, and these include dress suit or cilia for swimming, secretory glands for penetration into the host, a glycocalyx for host immuno-modulation or protection of the parasite, a gynaecophoric canal for continued coupling between sexes, and a well-organized reproductive organization for proper egg fertilization, as well as muscular suckers for feeding and attachment. This paper reviews these adaptation strategies, and how they are employed in survival. Additionally, possible interventions are proposed to counter the effect of these adaptations so as to make the parasites vulnerable to the hosts immune system and possible elimination.Adaptations for survival among schistosomes entail evasive strategies that enable the parasites to survive within the host without being affected by the hosts immunity. The evasion